Publications since:

2010

Li, X. (2010). (Invited Paper) Extending the maternal-zygotic effect with genomic imprinting.

Molecular Human Reproduction, DOI: 10.1093/molehr/gaq028.

2008

Li, X. *, Ito, M., Zhou, F., Youngson, N., Zuo, X., Leder, P. and Ferguson-Smith, A. (2008). A maternal-zygotic effect gene, Zfp57, maintains both maternal and paternal imprints. Developmental Cell 15, 547-557 (*, corresponding author)

2007

Li, X. * and Leder, P. * (2007). Identifying genes preferentially expressed in undifferentiated embryonic stem cells. BMC Cell Biology 8 (1): 37. (*, corresponding author)

2004

Chen,, J.,  Li, X. and Greenwald, I. (2004). sel-7, a positive regulator of lin-12 activity, encodes a novel nuclear protein in Caenorhabditis elegans. Genetics 166, 151-60.

2000

Wen,  C.,  Levitan, D. (*), Li, X. (*) and Greenwald, I. (2000). spr-2, a suppressor of the egg-laying defect caused by loss of sel-12 presenilin in Caenorhabditis elegans, is a member of the SET protein subfamily. Proc. Natl. Acad. Sci. USA 2000, 14524-9 (*, equal contribution).

1998

Nilsson, L., Li, X., Tiensuu, T.,  Auty, R., Greenwald I. and Tuck, S. (1998). C. elegans lin-25: cellular focus, protein expression and requirement for sur-2 during induction of vulval fates. Development 125, 4809-4819.

Li, X. and Greenwald, I. (1998). Additional evidence for an eight-transmembrane-domain topology for Caenorhabditis elegans  and human presenilins. Proc. Natl. Acad. Sci. USA 95, 7109-7114.

1997

Li, X. and Greenwald, I. (1997). HOP-1, a Caenorhabditis elegans presenilin, appears to be functionally redundant with SEL-12 presenilin and to facilitate LIN-12 and GLP-1 signaling. Proc. Natl. Acad. Sci. USA 94, 12204-12209.

1996

Li, X. and Greenwald, I. (1996). Membrane topology of the C. elegans SEL-12 presenilin. Neuron 17, 1015-1021.

© 2008 Mount Sinai Medical Center